Rheumatoid Arthritis Models

Rheumatoid Arthritis Models

Rheumatoid arthritis is a chronic autoimmune disorder characterized by persistent joint inflammation, leading to pain, swelling, and potential joint destruction. To facilitate the study of RA pathogenesis and the development of therapeutic interventions, we offers several well-established mouse models:

Model

CIA (Collagen-Induced Arthritis)

Strain

DBA

Induction

Type II collagen + CFA

Immune Response

Adaptive immunity (T & B cells, autoantibodies)

Key Features

Gradual, symmetrical polyarthritis; cartilage & bone erosion

Applications

Standard model for studying RA pathogenesis, testing DMARDs and biologics

Model

CAIA (Collagen Antibody-Induced Arthritis)

Strain

BALB/c

Induction

Anti-CII antibodies + LPS

Immune Response

Innate immunity (complement, neutrophils)

Key Features

Rapid onset; edema and joint destruction without prior sensitization

Applications

Fast evaluation of anti-inflammatory agents; studies on effector mechanisms

Model

AIA in Humanized Mice

Strain

huHSC-NCG-M

Induction

Adjuvant injection

Immune Response

Human immune cells (T cells, cytokines, macrophages)

Key Features

Systemic inflammation; human-like RA phenotype

Applications

Human-specific therapeutic evaluation; immunotherapy and cytokine studies

For more information:

Adjuvant-induced Arthritis (AIA) using HuHSC-NCG-M Humanized Mice

The Adjuvant-Induced Arthritis (AIA) model in HuHSC-NCG-M humanized mice is developed by administering Complete Freund’s Adjuvant (CFA) to induce systemic and joint-specific inflammation in mice. This model mimics human rheumatoid arthritis by activating human immune cells within a murine host, leading to joint swelling, synovial inflammation, and cartilage erosion.

Key Features:

  • Utilizes humanized mice with functional human T cells, B cells, and monocytes
  • Induces robust joint inflammation, resembling human RA pathology
  • Reflects both innate and adaptive human immune responses
  • Suitable for evaluating human-specific therapeutics such as monoclonal antibodies, cytokine inhibitors, and cell therapies
  • Enables investigation of human immune pathways, cytokine networks, and biomarker discovery in RA

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